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	<title>Tümör heterojenliği &#8211; BIOENGINEER.ORG</title>
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	<title>Tümör heterojenliği &#8211; BIOENGINEER.ORG</title>
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		<title>Polyclonal Origins of Early Human Colorectal Lesions</title>
		<link>https://bioengineer.org/polyclonal-origins-of-early-human-colorectal-lesions/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Tue, 25 Nov 2025 17:19:43 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[Erken kolorektal lezyonlar]]></category>
		<category><![CDATA[FAP ve kolorektal kanser]]></category>
		<category><![CDATA[İşte içeriğe uygun 5 etiket: **Poliklonal tümör kökeni]]></category>
		<category><![CDATA[Kanser paradigma kayması** * **Poliklonal tümör kökeni:** Makalenin ana bulgusu ve ana konusu olan]]></category>
		<category><![CDATA[Tümör heterojenliği]]></category>
		<category><![CDATA[tümörlerin tek bir hü]]></category>
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					<description><![CDATA[A Paradigm Shift in Cancer Origins: Revealing the Polyclonal Nature of Premalignant Colorectal Lesions Cancer research has long been dominated by the model that tumors arise from a single mutant cell which clonally expands to form a malignant mass. This classic monoclonal theory posits that one rogue cell acquires a sequence of genetic alterations conferring [&#8230;]]]></description>
		
		
		
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