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		<title>Splicing Factor LUC7L2 Emerges as a Driver of Kidney Damage from Chemotherapy</title>
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		<pubDate>Tue, 06 Oct 2026 04:53:33 +0000</pubDate>
				<category><![CDATA[Biology]]></category>
		<category><![CDATA[acute kidney injury]]></category>
		<category><![CDATA[alternative splicing]]></category>
		<category><![CDATA[cisplatin]]></category>
		<category><![CDATA[ferroptosis]]></category>
		<category><![CDATA[glutathione]]></category>
		<category><![CDATA[GPX4]]></category>
		<category><![CDATA[Lipid peroxidation]]></category>
		<category><![CDATA[LUC7L2]]></category>
		<category><![CDATA[p53]]></category>
		<category><![CDATA[proximal tubular cells]]></category>
		<category><![CDATA[single-cell RNA sequencing]]></category>
		<category><![CDATA[SLC7A11]]></category>
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					<description><![CDATA[New research reveals that the RNA splicing factor LUC7L2 drives ferroptotic kidney damage from cisplatin by corrupting the splicing of the antioxidant gene SLC7A11, exposing a potential biomarker and therapeutic target for chemotherapy-induced acute kidney injury.]]></description>
		
		
		
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