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		<title>Protein Clues Reveal Why Heart Risk Lingers After Bad Cholesterol Falls</title>
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				<category><![CDATA[Technology]]></category>
		<category><![CDATA[apolipoprotein B]]></category>
		<category><![CDATA[Atherosclerosis]]></category>
		<category><![CDATA[Cardiovascular disease]]></category>
		<category><![CDATA[European Heart Journal]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[LDL cholesterol]]></category>
		<category><![CDATA[Lipoprotein(a)]]></category>
		<category><![CDATA[Precision Medicine]]></category>
		<category><![CDATA[Proteomics]]></category>
		<category><![CDATA[residual risk]]></category>
		<category><![CDATA[triglyceride-rich lipoproteins]]></category>
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					<description><![CDATA[A large Swedish proteomics study of nearly 3,000 blood proteins in 35,000 UK Biobank participants reveals how triglyceride-rich lipoproteins and lipoprotein(a) drive inflammatory processes that explain heart disease risk remaining after LDL cholesterol is lowered.]]></description>
		
		
		
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