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		<title>How Broken Mitochondria Drive the Scarring That Slowly Kills the Kidneys</title>
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		<pubDate>Wed, 07 Oct 2026 18:43:22 +0000</pubDate>
				<category><![CDATA[Biology]]></category>
		<category><![CDATA[Chronic Kidney Disease]]></category>
		<category><![CDATA[Fatty Acid Oxidation]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[mitochondria]]></category>
		<category><![CDATA[Mitochondrial Dynamics]]></category>
		<category><![CDATA[Mitophagy]]></category>
		<category><![CDATA[mtDNA]]></category>
		<category><![CDATA[NAD+]]></category>
		<category><![CDATA[Renal fibrosis]]></category>
		<category><![CDATA[renal tubular epithelial cells]]></category>
		<category><![CDATA[Senescence]]></category>
		<category><![CDATA[single-cell transcriptomics]]></category>
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					<description><![CDATA[A sweeping new review argues that damaged mitochondria in kidney tubule cells act as both trigger and amplifier of the fibrotic scarring behind chronic kidney disease, mapping a path from organelle failure to whole-tissue remodeling.]]></description>
		
		
		
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