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	<title>Nöroenflamasyon &#8211; BIOENGINEER.ORG</title>
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	<title>Nöroenflamasyon &#8211; BIOENGINEER.ORG</title>
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		<title>Uncovering TIM-3’s Role in Alzheimer’s Microglia</title>
		<link>https://bioengineer.org/uncovering-tim-3s-role-in-alzheimers-microglia/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Mon, 26 Jan 2026 00:21:05 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[Alzheimer hastalığı]]></category>
		<category><![CDATA[İşte bu içerik için uygun 5 etiket: **TIM-3]]></category>
		<category><![CDATA[Mikroglia]]></category>
		<category><![CDATA[Nöroenflamasyon]]></category>
		<category><![CDATA[Tek Hücre Dizileme** **Açıklama:** 1. **TIM-3:** Çalışmanın ana odağı olan molekül. Araştırma bu reseptörün mikroglialardaki anormal ekspresyonunu ve]]></category>
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					<description><![CDATA[A groundbreaking study has recently emerged from the realm of neuroscience, providing significant insights into the evolving understanding of Alzheimer’s disease. The research, led by Xu et al., focuses on unraveling the complexities of microglial behavior during the progression of Alzheimer’s, specifically highlighting the aberrant expression of T-cell immunoglobulin and mucin domain 3 (TIM-3). Through [&#8230;]]]></description>
		
		
		
		<post-id xmlns="com-wordpress:feed-additions:1">320859</post-id>	</item>
		<item>
		<title>Delphinidin Eases Neuroinflammation, Behavior in Parkinson’s Mice</title>
		<link>https://bioengineer.org/delphinidin-eases-neuroinflammation-behavior-in-parkinsons-mice/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Mon, 12 Jan 2026 10:41:20 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[davranışsal iyileşme** **Açıklama:** 1. **delphinidin:** Çalışmanın ana odağı olan bileşik. 2. **Parkinson hastalığı:** Araştırmanın hedef aldığı temel]]></category>
		<category><![CDATA[İşte 5 uygun etiket: **delphinidin]]></category>
		<category><![CDATA[İşte 5 uygun etiket: `delphinidin]]></category>
		<category><![CDATA[mouse model]]></category>
		<category><![CDATA[Neuroinflammation]]></category>
		<category><![CDATA[Nöroenflamasyon]]></category>
		<category><![CDATA[nörokoruma]]></category>
		<category><![CDATA[Parkinson hastalığı]]></category>
		<category><![CDATA[Parkinson’s disease]]></category>
		<guid isPermaLink="false">https://bioengineer.org/delphinidin-eases-neuroinflammation-behavior-in-parkinsons-mice/</guid>

					<description><![CDATA[In a groundbreaking study set to redefine the therapeutic landscape for Parkinson’s disease (PD), researchers have unveiled the potent neuroprotective effects of delphinidin, a naturally occurring anthocyanidin found mainly in pigmented fruits and vegetables. This new research, recently published in npj Parkinson’s Disease, elucidates how delphinidin modulates neuroinflammation and alleviates behavioral deficits in a Parkinson’s [&#8230;]]]></description>
		
		
		
		<post-id xmlns="com-wordpress:feed-additions:1">316050</post-id>	</item>
		<item>
		<title>MicroRNA-199a-3p Enhances Neuroinflammation in Alzheimer’s Model</title>
		<link>https://bioengineer.org/microrna-199a-3p-enhances-neuroinflammation-in-alzheimers-model/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Sun, 30 Nov 2025 11:15:00 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[Alzheimer hastalığı]]></category>
		<category><![CDATA[MicroRNA-199a-3p]]></category>
		<category><![CDATA[mikroglia polarizasyonu]]></category>
		<category><![CDATA[Nöroenflamasyon]]></category>
		<category><![CDATA[transgenik fare modeli]]></category>
		<guid isPermaLink="false">https://bioengineer.org/microrna-199a-3p-enhances-neuroinflammation-in-alzheimers-model/</guid>

					<description><![CDATA[Research into the mechanisms behind Alzheimer’s disease (AD) has garnered increasing attention as the global population ages and the burden of neurological diseases escalates. A recent study has brought to light significant findings regarding the role of MicroRNA-199a-3p (miR-199a-3p) in modulating neuroinflammation within the context of Alzheimer’s pathology. Published in the esteemed journal BMC Neuroscience, [&#8230;]]]></description>
		
		
		
		<post-id xmlns="com-wordpress:feed-additions:1">303603</post-id>	</item>
		<item>
		<title>Unraveling NEC’s Impact on Premature Infant Brains</title>
		<link>https://bioengineer.org/unraveling-necs-impact-on-premature-infant-brains/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Fri, 21 Nov 2025 07:02:58 +0000</pubDate>
				<category><![CDATA[Technology]]></category>
		<category><![CDATA[bağırsak-beyin ekseni]]></category>
		<category><![CDATA[İşte içeriğe uygun 5 adet etiket: **NEC beyin hasarı]]></category>
		<category><![CDATA[NEC'in beyin üzerindeki y]]></category>
		<category><![CDATA[Nöroenflamasyon]]></category>
		<category><![CDATA[nörogelişimsel bozukluklar** **Açıklama:** 1. **NEC beyin hasarı:** Makalenin ana konusu]]></category>
		<category><![CDATA[Prematüre bebekler]]></category>
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					<description><![CDATA[In the ever-evolving landscape of neonatal medicine, a pervasive yet underrecognized threat continues to imperil premature infants: brain injury stemming from necrotizing enterocolitis (NEC). This complex and devastating condition, predominantly affecting preterm newborns, extends its impact far beyond the gut, unraveling profound neurological consequences that remain shrouded in clinical subtlety. Dr. D.J. Hackam’s groundbreaking research, [&#8230;]]]></description>
		
		
		
		<post-id xmlns="com-wordpress:feed-additions:1">299372</post-id>	</item>
		<item>
		<title>α-Synuclein Links Parkinson’s and Depression Pathways</title>
		<link>https://bioengineer.org/%ce%b1-synuclein-links-parkinsons-and-depression-pathways/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Thu, 20 Nov 2025 17:39:05 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[nörodejeneratif-psikiyatrik kesişim * **α-synuclein:** Metnin merkezinde bulunan ve Parkinson ile depresyon arasındaki bağın temelini oluşturan protein. * **Parkinson-depresyon kom]]></category>
		<category><![CDATA[Nöroenflamasyon]]></category>
		<category><![CDATA[nörotransmitter bozuklukları]]></category>
		<category><![CDATA[Parkinson-depresyon komorbiditesi]]></category>
		<category><![CDATA[α-synuclein]]></category>
		<guid isPermaLink="false">https://bioengineer.org/%ce%b1-synuclein-links-parkinsons-and-depression-pathways/</guid>

					<description><![CDATA[In a groundbreaking exploration of neurodegenerative and psychiatric intersections, recent research has illuminated the pivotal role of α-synuclein (α-syn) as a molecular nexus in Parkinson’s disease (PD) and depression. Often considered distinct entities, PD and depression are now emerging as intimately linked through converging pathogenic pathways, with α-synuclein accumulation serving as a central orchestrator of [&#8230;]]]></description>
		
		
		
		<post-id xmlns="com-wordpress:feed-additions:1">299253</post-id>	</item>
		<item>
		<title>Immune Gene Expression Patterns in Acute Stroke Unveiled</title>
		<link>https://bioengineer.org/immune-gene-expression-patterns-in-acute-stroke-unveiled/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Wed, 12 Nov 2025 05:48:35 +0000</pubDate>
				<category><![CDATA[Biology]]></category>
		<category><![CDATA[Akut İskemik İnme]]></category>
		<category><![CDATA[Bağışıklık Gen Ekspresyonu]]></category>
		<category><![CDATA[İmmün Gen Ekspresyonu]]></category>
		<category><![CDATA[İnme İmmünoterapisi **Seçilen Etiketlerin Gerekçesi:** 1. **Akut İskemik İnme (Acute Ischemic Stroke - AIS):** Araştırmanın temel konusu]]></category>
		<category><![CDATA[Nöroenflamasyon]]></category>
		<category><![CDATA[Prognostik Biyobelirteçler]]></category>
		<category><![CDATA[Terapötik Hedefler]]></category>
		<guid isPermaLink="false">https://bioengineer.org/immune-gene-expression-patterns-in-acute-stroke-unveiled/</guid>

					<description><![CDATA[Recent advances in neurological research have opened new avenues for understanding the underlying mechanisms of acute ischemic stroke (AIS). In a pivotal study, researchers Jin, Li, and Li have shed light on the dynamic expression patterns of immune-related genes in the peripheral blood of patients suffering from AIS. This groundbreaking work not only elucidates the [&#8230;]]]></description>
		
		
		
		<post-id xmlns="com-wordpress:feed-additions:1">295265</post-id>	</item>
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