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		<title>Atypical B Cells Emerge as Key Players in Gastric Cancer Spread, Study Finds</title>
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				<category><![CDATA[Health]]></category>
		<category><![CDATA[EMT]]></category>
		<category><![CDATA[gastric cancer]]></category>
		<category><![CDATA[Liver Metastases]]></category>
		<category><![CDATA[Metastasis]]></category>
		<category><![CDATA[MIF-CD74-CXCR4 axis]]></category>
		<category><![CDATA[Peritoneal metastases]]></category>
		<category><![CDATA[single-cell transcriptomics]]></category>
		<category><![CDATA[TAABs]]></category>
		<category><![CDATA[TNF-alpha signaling]]></category>
		<category><![CDATA[TP53 mutation]]></category>
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					<description><![CDATA[A large computational study links TTN/TP53 co-mutations in gastric cancer to a prognostically favorable population of tumor-associated atypical B cells that communicate with T cells through TNF-alpha signaling and may help explain why the disease spreads differently to the liver and peritoneum.]]></description>
		
		
		
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