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	<title>Lymphangioleiomyomatosis pathogenesis &#8211; BIOENGINEER.ORG</title>
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	<title>Lymphangioleiomyomatosis pathogenesis &#8211; BIOENGINEER.ORG</title>
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		<title>mTOR Dysregulation Drives Lung Repair Failure in LAM</title>
		<link>https://bioengineer.org/mtor-dysregulation-drives-lung-repair-failure-in-lam/</link>
		
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		<pubDate>Thu, 09 Oct 2025 15:51:33 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[Alveolar regeneration failure]]></category>
		<category><![CDATA[alveolar type 2 cell senescence]]></category>
		<category><![CDATA[IL-6 cytokine signaling]]></category>
		<category><![CDATA[IL-6 mediated cellular senescence]]></category>
		<category><![CDATA[lung repair mechanisms]]></category>
		<category><![CDATA[lymphangioleiomyomatosis (LAM)]]></category>
		<category><![CDATA[Lymphangioleiomyomatosis pathogenesis]]></category>
		<category><![CDATA[mTOR dysregulation]]></category>
		<category><![CDATA[mTOR signaling in pulmonary disease]]></category>
		<category><![CDATA[Senescence-targeted therapies for LAM]]></category>
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					<description><![CDATA[In a groundbreaking study poised to reshape our understanding of pulmonary diseases, researchers have uncovered a critical molecular mechanism underlying lung damage and impaired repair in lymphangioleiomyomatosis (LAM). The investigation, recently published in Nature Communications, elucidates how dysregulation of the mechanistic Target of Rapamycin (mTOR) pathway cascades into the release of the pro-inflammatory cytokine IL-6, [&#8230;]]]></description>
		
		
		
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