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	<title>H9c2 cardiomyocytes &#8211; BIOENGINEER.ORG</title>
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		<title>Body Clock Gene NPAS2 Shields the Diabetic Heart by Keeping Mitochondria Fueled</title>
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		<pubDate>Mon, 19 Oct 2026 07:19:48 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[AAV9 gene therapy]]></category>
		<category><![CDATA[cardiac fibrosis]]></category>
		<category><![CDATA[circadian clock]]></category>
		<category><![CDATA[diabetic cardiomyopathy]]></category>
		<category><![CDATA[glucose oxidation]]></category>
		<category><![CDATA[H9c2 cardiomyocytes]]></category>
		<category><![CDATA[lipotoxicity]]></category>
		<category><![CDATA[metabolic flexibility]]></category>
		<category><![CDATA[mitochondrial metabolism]]></category>
		<category><![CDATA[NPAS2]]></category>
		<category><![CDATA[oxidative stress]]></category>
		<category><![CDATA[pyruvate dehydrogenase]]></category>
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					<description><![CDATA[A new study shows that the circadian clock protein NPAS2 protects the diabetic heart by preserving pyruvate dehydrogenase activity and mitochondrial metabolic balance, suggesting a novel gene therapy target.]]></description>
		
		
		
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