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	<title>Fibroblast-kanser hücresi iletişimi** * **Meme kanseri kemoterapi direnci:** Çalışmanın odaklandığı temel klinik sor &#8211; BIOENGINEER.ORG</title>
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	<title>Fibroblast-kanser hücresi iletişimi** * **Meme kanseri kemoterapi direnci:** Çalışmanın odaklandığı temel klinik sor &#8211; BIOENGINEER.ORG</title>
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		<title>NRG1/PDGFC Loop Fuels Breast Cancer Drug Resistance</title>
		<link>https://bioengineer.org/nrg1-pdgfc-loop-fuels-breast-cancer-drug-resistance/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Mon, 10 Nov 2025 22:08:23 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[Ferroptozis inhibisyonu]]></category>
		<category><![CDATA[Fibroblast-kanser hücresi iletişimi** * **Meme kanseri kemoterapi direnci:** Çalışmanın odaklandığı temel klinik sor]]></category>
		<category><![CDATA[İçeriğe uygun 5 etiket: **Meme kanseri kemoterapi direnci]]></category>
		<category><![CDATA[NRG1/PDGFC sinyal aksı]]></category>
		<category><![CDATA[Tümör mikroçevresi]]></category>
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					<description><![CDATA[In a groundbreaking study poised to redefine therapeutic approaches in oncology, researchers have unveiled a novel tumor microenvironment interaction that critically governs treatment resistance in breast cancer. This discovery elucidates a complex biochemical dialogue between fibroblasts and cancer cells mediated through the NRG1/PDGFC signaling axis, which fortifies breast cancer cells against the chemotherapeutic agent paclitaxel. [&#8230;]]]></description>
		
		
		
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