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		<title>Epithelial Cells&#8217; Sensitivity to Interferon Gamma May Shape Genital Herpes Outcomes</title>
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		<pubDate>Thu, 08 Oct 2026 14:50:59 +0000</pubDate>
				<category><![CDATA[Biology]]></category>
		<category><![CDATA[Health]]></category>
		<category><![CDATA[antiviral defense]]></category>
		<category><![CDATA[chemokines]]></category>
		<category><![CDATA[CXCL10]]></category>
		<category><![CDATA[epithelial immunity]]></category>
		<category><![CDATA[genital herpes]]></category>
		<category><![CDATA[HSV-2]]></category>
		<category><![CDATA[interferon-gamma]]></category>
		<category><![CDATA[keratinocytes]]></category>
		<category><![CDATA[PLOS Pathogens]]></category>
		<category><![CDATA[STAT1]]></category>
		<category><![CDATA[tissue-resident T cells]]></category>
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					<description><![CDATA[New research in PLOS Pathogens shows that genital epithelial cells from people with asymptomatic HSV-2 infection respond more strongly to interferon gamma, producing more of the immune-recruiting chemokine CXCL10 and potentially explaining why some carriers never develop lesions.]]></description>
		
		
		
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