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		<title>REV-ERBβ emerges as a non-redundant brake on TH17-driven inflammation</title>
		<link>https://bioengineer.org/rev-erb%ce%b2-emerges-as-a-non-redundant-brake-on-th17-driven-inflammation/</link>
		
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		<pubDate>Sat, 17 Oct 2026 16:04:28 +0000</pubDate>
				<category><![CDATA[Technology]]></category>
		<category><![CDATA[autoimmunity]]></category>
		<category><![CDATA[circadian clock]]></category>
		<category><![CDATA[colitis]]></category>
		<category><![CDATA[EAE]]></category>
		<category><![CDATA[heme]]></category>
		<category><![CDATA[IL-17A]]></category>
		<category><![CDATA[inflammation]]></category>
		<category><![CDATA[NCoR1]]></category>
		<category><![CDATA[nuclear receptor]]></category>
		<category><![CDATA[REV-ERBα]]></category>
		<category><![CDATA[REV-ERBβ]]></category>
		<category><![CDATA[Th17 cells]]></category>
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					<description><![CDATA[New research reveals that the circadian nuclear receptor REV-ERBβ acts as a distinct, non-redundant negative regulator of pro-inflammatory TH17 cell responses, independent of heme and the co-repressor NCoR1.]]></description>
		
		
		
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