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		<title>Mitochondria’s Role in PANoptosis: Mechanisms and Therapies</title>
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		<pubDate>Tue, 21 Oct 2025 22:28:29 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[apoptosis-inflammation crosstalk]]></category>
		<category><![CDATA[disease-specific molecular targets]]></category>
		<category><![CDATA[mitochondria-targeted therapies]]></category>
		<category><![CDATA[Mitochondrial apoptosis mechanisms]]></category>
		<category><![CDATA[mitochondrial dynamics in disease]]></category>
		<category><![CDATA[Mitochondrial dysfunction in disease]]></category>
		<category><![CDATA[mitochondrial outer membrane permeabilization]]></category>
		<category><![CDATA[PANoptosis pathways]]></category>
		<category><![CDATA[Reactive oxygen species signaling]]></category>
		<category><![CDATA[Therapeutic targeting of mitochondria]]></category>
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					<description><![CDATA[Mitochondria, long recognized as the powerhouse of the cell, now emerge as pivotal regulators in the intrinsic apoptosis pathway, orchestrating decisive cellular fate determinations. Central to this function is their ability to mediate mitochondrial outer membrane permeabilization (MOMP), which initiates a cascade involving the release of cytochrome c (Cyt c), mitochondrial reactive oxygen species (mtROS), [&#8230;]]]></description>
		
		
		
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