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	<title>cytokine crosstalk &#8211; BIOENGINEER.ORG</title>
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		<title>Rival immune messengers turn out to be secret partners in healing</title>
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				<category><![CDATA[Health]]></category>
		<category><![CDATA[cytokine crosstalk]]></category>
		<category><![CDATA[gene expression]]></category>
		<category><![CDATA[H3K27me3]]></category>
		<category><![CDATA[IL-4]]></category>
		<category><![CDATA[immunology]]></category>
		<category><![CDATA[IRF1]]></category>
		<category><![CDATA[macrophages]]></category>
		<category><![CDATA[monocytes]]></category>
		<category><![CDATA[NF-kB]]></category>
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		<category><![CDATA[TNF]]></category>
		<category><![CDATA[Wound Healing]]></category>
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					<description><![CDATA[A Nature Communications study shows that the proinflammatory cytokine TNF acts mainly as a costimulator of IL-4-driven gene expression in human monocytes and macrophages, while IL-4 selectively inhibits only the TNF-induced interferon response through chromatin-level mechanisms.]]></description>
		
		
		
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