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	<title>chemotherapy-induced cardiomyopathy &#8211; BIOENGINEER.ORG</title>
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	<title>chemotherapy-induced cardiomyopathy &#8211; BIOENGINEER.ORG</title>
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		<title>New Study in Chinese Medical Journal Uncovers GSTP1’s Protective Role Against Ferroptosis and Doxorubicin-Induced Cardiac Damage</title>
		<link>https://bioengineer.org/new-study-in-chinese-medical-journal-uncovers-gstp1s-protective-role-against-ferroptosis-and-doxorubicin-induced-cardiac-damage/</link>
		
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		<pubDate>Wed, 05 Nov 2025 18:27:02 +0000</pubDate>
				<category><![CDATA[Cancer]]></category>
		<category><![CDATA[cardioprotective molecular mechanisms]]></category>
		<category><![CDATA[chemotherapy-induced cardiomyopathy]]></category>
		<category><![CDATA[doxorubicin cardiotoxicity]]></category>
		<category><![CDATA[GSTP1 ferroptosis protection]]></category>
		<category><![CDATA[JNK pathway ACSL4 regulation]]></category>
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					<description><![CDATA[In a groundbreaking study poised to reshape our understanding of chemotherapy-induced cardiac injury, researchers from West China Hospital of Sichuan University have identified a novel molecular pathway linking doxorubicin toxicity with ferroptosis, a regulated cell death modality pivotal in cardiomyocyte damage. This discovery elucidates how doxorubicin (DOX), a widely used anthracycline chemotherapeutic agent, exerts deleterious [&#8230;]]]></description>
		
		
		
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