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		<title>Metabolic Switch in Heart Cells Rewinds Injury Into Regeneration, Study Finds</title>
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		<pubDate>Thu, 08 Oct 2026 21:42:09 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[cardiac regeneration]]></category>
		<category><![CDATA[cardiomyocyte proliferation]]></category>
		<category><![CDATA[epigenetics]]></category>
		<category><![CDATA[Fibrosis]]></category>
		<category><![CDATA[H3K27me3]]></category>
		<category><![CDATA[H3K4me3]]></category>
		<category><![CDATA[malonate]]></category>
		<category><![CDATA[metabolism]]></category>
		<category><![CDATA[Myocardial Infarction]]></category>
		<category><![CDATA[myofibroblasts]]></category>
		<category><![CDATA[Single-nucleus sequencing]]></category>
		<category><![CDATA[succinate dehydrogenase]]></category>
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					<description><![CDATA[New research shows that transient inhibition of the mitochondrial enzyme succinate dehydrogenase triggers coordinated metabolic and epigenetic reprogramming across heart cell types, reactivating regenerative programs and suppressing fibrosis after myocardial infarction.]]></description>
		
		
		
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