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	<title>cancer cell proliferation &#8211; BIOENGINEER.ORG</title>
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		<title>Aurora-A Boosts HCC Growth by Regulating Mitochondria</title>
		<link>https://bioengineer.org/aurora-a-boosts-hcc-growth-by-regulating-mitochondria/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Sat, 13 Dec 2025 01:44:02 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[Aurora-A kinase]]></category>
		<category><![CDATA[cancer cell proliferation]]></category>
		<category><![CDATA[hepatocellular carcinoma (HCC)]]></category>
		<category><![CDATA[Maf1 transcriptional regulation]]></category>
		<category><![CDATA[mitochondrial regulation]]></category>
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					<description><![CDATA[In an unprecedented breakthrough, researchers have uncovered a novel molecular pathway that significantly advances our understanding of hepatocellular carcinoma (HCC), a formidable type of liver cancer. The study reveals that Aurora-A kinase influences the subcellular localization of the transcriptional regulator Maf1, driving cancer cell proliferation by modulating mitochondrial function. This insight not only charts new [&#8230;]]]></description>
		
		
		
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		<title>FAM111B Knockdown Suppresses Ovarian Cancer by Downregulating MYC</title>
		<link>https://bioengineer.org/fam111b-knockdown-suppresses-ovarian-cancer-by-downregulating-myc/</link>
		
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		<pubDate>Sat, 09 Aug 2025 03:00:43 +0000</pubDate>
				<category><![CDATA[Cancer]]></category>
		<category><![CDATA[cancer cell proliferation]]></category>
		<category><![CDATA[FAM111B gene]]></category>
		<category><![CDATA[MYC oncogene]]></category>
		<category><![CDATA[ovarian cancer therapy]]></category>
		<category><![CDATA[tumorigenesis suppression]]></category>
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					<description><![CDATA[In a groundbreaking study published in BMC Cancer, researchers have unveiled a pivotal molecular mechanism that could reshape therapeutic approaches to ovarian cancer—a disease notoriously challenging both in diagnosis and treatment due to its aggressive nature and high mortality rate. The study zeroes in on FAM111B, a gene whose functional role in ovarian cancer has [&#8230;]]]></description>
		
		
		
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