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		<title>Cell Division Switch in Pseudomonas Reveals New Route to Antibiotic Resistance</title>
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				<category><![CDATA[Biology]]></category>
		<category><![CDATA[antibiotic resistance]]></category>
		<category><![CDATA[aztreonam]]></category>
		<category><![CDATA[beta-lactam antibiotics]]></category>
		<category><![CDATA[cell division]]></category>
		<category><![CDATA[divisome]]></category>
		<category><![CDATA[FtsI]]></category>
		<category><![CDATA[FtsN]]></category>
		<category><![CDATA[FtsQLB complex]]></category>
		<category><![CDATA[pedestal domain]]></category>
		<category><![CDATA[peptidoglycan synthesis]]></category>
		<category><![CDATA[Pseudomonas aeruginosa]]></category>
		<category><![CDATA[SEDS-bPBP synthase]]></category>
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					<description><![CDATA[Mutations in the pedestal domain of the cell division protein FtsI alter how Pseudomonas aeruginosa regulates septal cell wall synthesis and can either increase or decrease its susceptibility to β-lactam antibiotics.]]></description>
		
		
		
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