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		<title>Lens Capsule Proteomics Reveals Lysosomal Breakdown Linking Cataract and Glaucoma</title>
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		<pubDate>Tue, 06 Oct 2026 01:36:33 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[age-related cataract]]></category>
		<category><![CDATA[anterior lens capsule]]></category>
		<category><![CDATA[ATP6V1A]]></category>
		<category><![CDATA[intraocular pressure]]></category>
		<category><![CDATA[lens epithelial cells]]></category>
		<category><![CDATA[lysosomal dysfunction]]></category>
		<category><![CDATA[mass spectrometry]]></category>
		<category><![CDATA[primary angle-closure glaucoma]]></category>
		<category><![CDATA[Proteomics]]></category>
		<category><![CDATA[proteostasis]]></category>
		<category><![CDATA[UK Biobank]]></category>
		<category><![CDATA[v-ATPase]]></category>
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					<description><![CDATA[Proteomic analysis of human anterior lens capsules reveals that ATP6V1A-associated lysosomal dysfunction links elevated eye pressure and lens epithelial injury in patients with both age-related cataract and primary angle-closure glaucoma.]]></description>
		
		
		
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