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		<title>Infections May Not Trigger Graves&#8217; Disease, Genetic Study of Antibody Responses Suggests</title>
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		<pubDate>Fri, 16 Oct 2026 19:38:52 +0000</pubDate>
				<category><![CDATA[Biology]]></category>
		<category><![CDATA[Health]]></category>
		<category><![CDATA[antibody responses]]></category>
		<category><![CDATA[autoimmune thyroid disease]]></category>
		<category><![CDATA[cytomegalovirus]]></category>
		<category><![CDATA[Epstein-Barr virus]]></category>
		<category><![CDATA[Genome-wide association study]]></category>
		<category><![CDATA[Graves' disease]]></category>
		<category><![CDATA[Helicobacter pylori]]></category>
		<category><![CDATA[humoral immunity]]></category>
		<category><![CDATA[Mendelian randomization]]></category>
		<category><![CDATA[molecular mimicry]]></category>
		<category><![CDATA[polyomavirus]]></category>
		<category><![CDATA[thyroid autoimmunity]]></category>
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					<description><![CDATA[A bidirectional Mendelian randomization study finds no evidence that antibody responses to infections cause Graves' disease, instead linking genetic liability to the condition with altered antibody levels against cytomegalovirus, Helicobacter pylori, and polyomaviruses.]]></description>
		
		
		
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