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	<title>anti-fibrotic therapies &#8211; BIOENGINEER.ORG</title>
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		<title>Diseased Blood Vessels May Drive Fibrosis Across Multiple Organs</title>
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		<pubDate>Tue, 27 Oct 2026 17:59:42 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[angiocrine signaling]]></category>
		<category><![CDATA[angiogenesis]]></category>
		<category><![CDATA[anti-fibrotic therapies]]></category>
		<category><![CDATA[capillary de-specialization]]></category>
		<category><![CDATA[Endothelial Cells]]></category>
		<category><![CDATA[Fibroblast Activation]]></category>
		<category><![CDATA[Fibrosis]]></category>
		<category><![CDATA[hypoxia]]></category>
		<category><![CDATA[review article]]></category>
		<category><![CDATA[vascular]]></category>
		<category><![CDATA[Vascular aging]]></category>
		<category><![CDATA[vascular dysfunction]]></category>
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					<description><![CDATA[A new review argues that dysfunctional blood vessels, through failed endothelial quiescence, abnormal remodeling modes and disrupted angiocrine signaling, may orchestrate fibrotic progression across multiple organs.]]></description>
		
		
		
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