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	<title>acute lung injury &#8211; BIOENGINEER.ORG</title>
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		<title>IL-1β+ Lung Macrophages Drive Sepsis Lung Injury</title>
		<link>https://bioengineer.org/il-1%ce%b2-lung-macrophages-drive-sepsis-lung-injury/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Mon, 08 Dec 2025 21:35:11 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[acute lung injury]]></category>
		<category><![CDATA[Endothelial dysfunction]]></category>
		<category><![CDATA[IL-1β macrophages]]></category>
		<category><![CDATA[Immune-metabolic crosstalk]]></category>
		<category><![CDATA[Sepsis-induced lung injury]]></category>
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					<description><![CDATA[In a groundbreaking study that promises to reshape our understanding of sepsis-induced lung injury, researchers have unveiled the critical role of IL-1β positive lung-resident macrophages in mediating endothelial dysfunction. This discovery shines a spotlight on the complex immune-metabolic interplay that drives acute lung injury (ALI) in sepsis, a condition notorious for its high mortality rates [&#8230;]]]></description>
		
		
		
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		<title>N6-Methyladenosine Methylation Identified as a Promising Therapeutic Target for Acute Lung Injury</title>
		<link>https://bioengineer.org/n6-methyladenosine-methylation-identified-as-a-promising-therapeutic-target-for-acute-lung-injury/</link>
		
		<dc:creator><![CDATA[Bioengineer]]></dc:creator>
		<pubDate>Thu, 30 Oct 2025 11:24:32 +0000</pubDate>
				<category><![CDATA[Health]]></category>
		<category><![CDATA[acute lung injury]]></category>
		<category><![CDATA[epigenetic therapy]]></category>
		<category><![CDATA[m6A methylation]]></category>
		<category><![CDATA[pulmonary inflammation]]></category>
		<category><![CDATA[RNA metabolism]]></category>
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					<description><![CDATA[Acute Lung Injury (ALI) remains a formidable challenge in critical care medicine, marked by devastating inflammation of the lung parenchyma and persistent hypoxemia. As mortality rates remain high, unraveling the molecular drivers of this pathological condition is imperative for the development of effective therapies. Recent advances have spotlighted the epigenetic regulation of gene expression, particularly [&#8230;]]]></description>
		
		
		
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